Figures 61, 62, 63: A, Physeal fracture with posterior angulation of the epiphysis. B, Spiral fracture. There is also an old malunion of the lateral humeral condyle. C, Comminuted fracture

Figures 64, 65: Osteomyelitis due to coccidiodes immitis infection. The multicentric cres of involvement are manifested as zone of sclerotic bone proliferation. Left, One of the areas of osteomyelitis crossed the joint space at the elbow, which helps to differentiate this condition from osteogenic sarcoma. The multicentric origin would also assist in differentiating this condition from osteogenic sarcoma.

Figures 66: Osteomyelitis due to blastomycosis. The lesion is mainly lytic with only a slight suggestion of a sclerotic halo.

Figures 67, 68: Osteomyelitis of the distal ulna of a lion due to a bite wound from a mountain lion. In many respects this lesion resembels an osteogenic sarcoma. Biopsy may be the only method of making a positive diagnosis in a case such as these.

Figures 69, 70: Osteomyelitis in the ulna of a cat due to a bite wound (arrow). In many respects, this lesion resembles an osteogenic sarcoma. Biopsy and/or culture may be the only method of making a positive diagnosis.

Figures 71, 72: Hypertrophic pulmonary osteopathy involving all of the long bones is shown in left and right. The thoracic lesion was a metastatic interductile mammary adenocarcinima.

Figure 73: Osteochondroma involving the distal ulna. This lesion has enlarged to such an extent that radius is remodeling in the area of contact.

Figure 74: Benign bone tumor of the fourth metatarsal none in a one-year-old female collie. Most likely this represents an enchondroma. All the radiographic signs of a benign or nonregressive bone lesion are present. Note the medial deviation of the third metacarpal bone due to the expansion of the primary lesion.

Figure 75: Generalized osteoporosis is evident in this skeleton of an immature cat. Cortical thinness and evidence of pathologic fracture are apparent.

Figure 76: Capital epiphyseal separation on the right, resulting in avascular necrosis of the right femoral neck. The neck of the affected femur is being resorbed due to the hyperimia associated with the fracture. The avascular necrotic femoral head is unchanged in shape. since the blood supply to the femoral head is completely disrupted, the necrotic bone cannot be removed.

Figure 77: Aseptic necrosis of the femoral heads in the healed stage.

Figures 78, 79: Degenerative joint disease of the stifle. The predominebt lesion is periarticular osteophyte formation. The joint space is of normal width. Areas suggestive of subchondral cysts are noted in the proximal tibia.